Organ specific autoimmune diseases
Bacteria are extremely versatile microorganisms that continuously sense the gastrointestinal environmental conditions and can rapidly alter their behavior and appearance to survive and reproduce. This process enables the bacteria to hide without provoking an inflammatory response from the immune system.
The measurement of multiple cytokines will present the CAM practitioner with a broader picture of the patient’s inflammatory condition, and thereby would be useful in fitting a therapeutic intervention on an individual basis. - Vojani
A common method of evasion is suppression of pro-inflammatory cytokines. The microbes target those cells of the immune system that specifically react against them. By disabling or suppressing those cells, the microbes prevent the body from mounting an immune response. The Endotoxin lipopolysaccharide (LPS) causes a reduced ability to produce pro-inflammatory cytokines. Exposure to LPS influences cytokine production resulting in a shift toward a TH2 cytokine response when bacteria should cause a TH1 response. This would cause mixed results for those doing a TH1/TH2 Challenge. Many of the NEI Stimulated Cytokine test results showed a mixed TH2 and TH17 immune response.
Small intestinal bacterial overgrowth (SIBO)
Small Intestinal Bacterial Overgrowth (SIBO), also termed bacterial overgrowth, is a disorder of excessive bacterial growth in the small intestine. When bacteria migrate into the upper sections of the small intestine, they are no longer beneficial becoming detrimental to health. SIBO bacteria produce more toxic substances some of which alter hormone elimination.
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When SIBO occurs, Clostridia species of bacteria convert bile salts into toxic bile salts. Toxic secondary bile acid levels are damaging to the cells lining the small intestine and cause extensive mucosal damage of the stomach and esophagus. This is more harmful than acid reﬂux alone. The major conversion of secondary bile salts is the formation of lithocholic acid. Lithocholic acid competitively inhibits etiocholanolone elimination occurring in the liver. This results in increase potential for immune stimulation and a localized fever. In addition to this lithocholic acid causes damage to Vitamin D receptors and reactions to sunlight or Vitamin D supplments.
Etiocholanolone is a little known hormone that is produced just after DHEA. Actually etiocholanolone is produced from androstenedione. Etiocholanolone is responsible for producing short-term inflammation with symptoms of chills, anorexia, headache, malaise, muscle pain, joint pain, and inflammatory responses.
Excessive DHEA supplementation may be the cause of high etiocholanolone levels. Certain individuals have episodic bouts of inflammation or fever due to periodic accumulation in the blood of unprocessed Etiocholanolone. Some may describe these episodic bouts as hot flashes.
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Fuck you, judgemental piece of shitby 201019812007
I don't train poorly - I'm just accident prone and there's a physiological reason why I've torn both hip labrums and both shoulder labrums - I have multiple autoimmune diseases and am hypersensitive to things... I have a major overgrowth of cartilage in and around many of my articulations... pair that with my active lifestyle and you've got disaster and multiple injuries.
now I could bitch and moan about it the way you bitch and moan about everything, but I choose not to let it stop me or slow me down so fuck you very much, asshat.
I have a paper on adrenal insufficiency ifby samshine
You would like to take a look. Too long to post here, but I could email it.
This was from a canine health symposium I attended earlier this year.
"Which dogs should be tested for adrenal exhaustion? Those with: chronic allergies, infections,
IBD, pancreatitis; Cushingoid signs but negative Cushings tests, SARDS, poorly controlled
diabetes or epilepsy, multiple autoimmune diseases."
She said if you have a dog that keeps developing allergies, check into this.
This lady also has a couple books out.
Clinical and Experimental Studies of Organ-Specific Autoimmune Diseases: With Special Reference to Addison's Diease and Autoimmune Hepatitus ... from the Faculty of Medicine, 1045)
Book (Uppsala Universitet)
Vaccine Induced Immune Overload and the Epidemic of Chronic Autoimmune .. — The People's Voice
Family studies indicate type 1 diabetes is linked to the development of several different autoimmune diseases including organ specific autoimmune diseases and rheumatoid diseases.
Autoimmune Diseases and Treatment: Organ-Specific and Systemic Disorders (Annals of the New York Academy of Sciences)
Organ-Specific Autoantibodies in Addison's Disease & Autoimmune Polyendocrine Syndrome Type I (Comprehensive Summaries of Uppsala Dissertations from the Faculty of Medicine, 944)
Book (Uppsala Universitet)
Endocrine and Organ Specific Autoimmunity (Molecular Biology Intelligence Unit)
Book (R G Landes Co)
Hematology: A Pathophysiologic Approach (Mosby's Physiology Monograph)